Atıf İçin Kopyala
Ozkaya M., Baykan A., Cakir M., Vural C., Sunkak S., Unal E., ...Daha Fazla
INTERNATIONAL JOURNAL OF CARDIOLOGY, cilt.333, ss.174-183, 2021 (SCI-Expanded)
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Yayın Türü:
Makale / Tam Makale
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Cilt numarası:
333
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Basım Tarihi:
2021
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Doi Numarası:
10.1016/j.ijcard.2021.02.020
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Dergi Adı:
INTERNATIONAL JOURNAL OF CARDIOLOGY
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Derginin Tarandığı İndeksler:
Science Citation Index Expanded (SCI-EXPANDED), Scopus, Academic Search Premier, PASCAL, BIOSIS, CAB Abstracts, EMBASE, MEDLINE, Public Affairs Index, Veterinary Science Database
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Sayfa Sayıları:
ss.174-183
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Anahtar Kelimeler:
Acute rheumatic fever, ARF, Mucosal-associated invariant cell, MAIT, INVARIANT T-CELLS, HEART-DISEASE, MAIT CELLS, CYTOKINES, AUTOIMMUNITY, PATHOGENESIS, PROGRESSION, STATEMENT, CLONES, IL-17
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Erciyes Üniversitesi Adresli:
Evet
Özet
Background: Acute rheumatic fever (ARF) is an autoimmune disease caused by group A beta-hemolytic streptococci (GAS) and may develop into rheumatic heart disease (RHD). The pathogenesis of ARF and RHD involves molecular mimicry and antibody-mediated mechanisms. T cell involvement is described in various stages of the disease. Mucosal associated invariant T (MATT) cells are enriched at the mucosa and are present in the blood and may be activated by GAS.