A Different View on the Etiopathogenesis of Attention-deficit Hyperactivity Disorder from an Inflammation Perspective

DURSUN S., DEMİRCİ E., KILIÇ E., ÖZMEN S.

CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE, cilt.19, sa.1, ss.145-154, 2021 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 19 Sayı: 1
  • Basım Tarihi: 2021
  • Doi Numarası: 10.9758/cpn.2021.19.1.145
  • Dergi Adı: CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus, Academic Search Premier, BIOSIS, EMBASE, Psycinfo
  • Sayfa Sayıları: ss.145-154
  • Anahtar Kelimeler: Attention deficit disorder with hyperactivity, Interleukin-1 beta, Interleukin-6, Inflammation, Tumor necrosis factor-alpha, Visfatin, SERUM VISFATIN LEVELS, DEFICIT/HYPERACTIVITY DISORDER, EATING-DISORDERS, CYTOKINE, INTERLEUKIN-1, ASSOCIATION, CORTISOL, NERVOSA, ADHD, IL-6
  • Erciyes Üniversitesi Adresli: Evet

Özet

Objective: Attention-deficit hyperactivity disorder (ADHD) has a complex etiology and genetic, environmental and biological factors are considered to play a role in the etiology of ADHD by mutually interacting. Recent studies have emphasized that inflammation may be present in the etiology of ADHD. This study aims to investigate the possible role of visfatin, IL-6, IL-1 beta and TNF-alpha molecules in the etiology of ADHD.