A Different View on the Etiopathogenesis of Attention-deficit Hyperactivity Disorder from an Inflammation Perspective

DURSUN S., DEMİRCİ E., KILIÇ E., ÖZMEN S.

CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE, vol.19, no.1, pp.145-154, 2021 (SCI-Expanded) identifier identifier identifier

  • Publication Type: Article / Article
  • Volume: 19 Issue: 1
  • Publication Date: 2021
  • Doi Number: 10.9758/cpn.2021.19.1.145
  • Journal Name: CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE
  • Journal Indexes: Science Citation Index Expanded (SCI-EXPANDED), Scopus, Academic Search Premier, BIOSIS, EMBASE, Psycinfo
  • Page Numbers: pp.145-154
  • Keywords: Attention deficit disorder with hyperactivity, Interleukin-1 beta, Interleukin-6, Inflammation, Tumor necrosis factor-alpha, Visfatin, SERUM VISFATIN LEVELS, DEFICIT/HYPERACTIVITY DISORDER, EATING-DISORDERS, CYTOKINE, INTERLEUKIN-1, ASSOCIATION, CORTISOL, NERVOSA, ADHD, IL-6
  • Erciyes University Affiliated: Yes

Abstract

Objective: Attention-deficit hyperactivity disorder (ADHD) has a complex etiology and genetic, environmental and biological factors are considered to play a role in the etiology of ADHD by mutually interacting. Recent studies have emphasized that inflammation may be present in the etiology of ADHD. This study aims to investigate the possible role of visfatin, IL-6, IL-1 beta and TNF-alpha molecules in the etiology of ADHD.