CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE, vol.19, no.1, pp.145-154, 2021 (SCI-Expanded)
Article / Article
CLINICAL PSYCHOPHARMACOLOGY AND NEUROSCIENCE
Science Citation Index Expanded (SCI-EXPANDED), Scopus, Academic Search Premier, BIOSIS, EMBASE, Psycinfo
Attention deficit disorder with hyperactivity, Interleukin-1 beta, Interleukin-6, Inflammation, Tumor necrosis factor-alpha, Visfatin, SERUM VISFATIN LEVELS, DEFICIT/HYPERACTIVITY DISORDER, EATING-DISORDERS, CYTOKINE, INTERLEUKIN-1, ASSOCIATION, CORTISOL, NERVOSA, ADHD, IL-6
Erciyes University Affiliated:
Objective: Attention-deficit hyperactivity disorder (ADHD) has a complex etiology and genetic, environmental and biological factors are considered to play a role in the etiology of ADHD by mutually interacting. Recent studies have emphasized that inflammation may be present in the etiology of ADHD. This study aims to investigate the possible role of visfatin, IL-6, IL-1 beta and TNF-alpha molecules in the etiology of ADHD.