Benter, I. Et Al. 2005. Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery. PHARMACOLOGICAL RESEARCH , vol.52, no.3 , 252-257.

Benter, I., Yousif, M., Canatan, H., & Akhtar, S., (2005). Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery. PHARMACOLOGICAL RESEARCH , vol.52, no.3, 252-257.

Benter, IF Et Al. "Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery," PHARMACOLOGICAL RESEARCH , vol.52, no.3, 252-257, 2005

Benter, IF Et Al. "Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery." PHARMACOLOGICAL RESEARCH , vol.52, no.3, pp.252-257, 2005

Benter, I. Et Al. (2005) . "Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery." PHARMACOLOGICAL RESEARCH , vol.52, no.3, pp.252-257.

@article{article, author={IF Benter Et Al. }, title={Inhibition of Ca2+/calmodulin-dependent protein kinase II, RAS-GTPase and 20-hydroxyeicosatetraenoic acid attenuates the development of diabetes-induced vascular dysfunction in the rat carotid artery}, journal={PHARMACOLOGICAL RESEARCH}, year=2005, pages={252-257} }